The AGA Treatment Revolution 43

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On the other hand, it is believed that the primary gene determining the sensitivity of androgen receptors, which capture dihydrotestosterone, is located on the X chromosome.

A son born with an XY pair inherits the X chromosome from his mother and the Y chromosome from his father. Since the gene determining the sensitivity of androgen receptors is on the X chromosome, whether a son is prone to male pattern baldness is determined not by the father's but by the mother's genetics.

If the mother's father had a strong sensitivity to androgen receptors, there is a 50% chance that the son will also have a strong sensitivity, making him more susceptible to male pattern baldness.

The reason why a mother might not experience male pattern baldness is due to her being female, and it is considered that her son inherits the risk of thinning hair. This explains why it's often said that if a grandfather had thinning hair and his grandson has it too, it's a case of "skipping a generation" in genetics.

Of course, thinning hair is not solely determined by genetics. Environmental factors also play a role.

Maintaining a balanced diet with essential nutrients for hair growth and mitigating stress through moderate exercise should not be overlooked. These points are elaborated on in Chapter 6, which you may refer to for more information.

Most importantly, it's about being proactive. If you suspect a high genetic risk for male pattern baldness based on your grandfather's scalp condition or genetic testing, start using medications like finasteride or minoxidil even before hair loss becomes noticeable.

As far as I know, most male doctors specializing in male pattern baldness who are aware of their genetic predisposition start using finasteride and minoxidil promptly.

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